<i>S</i>-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability.

导出 OA

翻译打印收藏纠错

第一作者： Patricia,Zamorano

第一单位： Instituto de Inmunología, Facultad de Medicina, Universidad Austral de Chile, Valdivia, Chile.

作者： Patricia,Zamorano ^[1] ; Natalie,Marín ^[1] ; Francisco,Córdova ^[1] ; Alejandra,Aguilar ^[1] ; Cynthia,Meininger ^[2] ; Mauricio P,Boric ^[3] ; Nikola,Golenhofen ^[4] ; Jorge E,Contreras ^[5] ; José,Sarmiento ^[6] ; Walter N,Durán ^[5] ; Fabiola A,Sánchez ^[7]

作者单位： Instituto de Inmunología, Facultad de Medicina, Universidad Austral de Chile, Valdivia, Chile. ^[1] Department of Medical Physiology, Texas A&M Health Science Center, Temple, Texas. ^[2] Departamento de Fisiología, Facultad de Ciencias Biológicas, Pontificia Universidad Católica de Chile, Santiago, Chile. ^[3] Institute of Anatomy and Cell Biology, University of Ulm, Ulm, Germany; and. ^[4] Department of Pharmacology, Physiology and Neuroscience, New Jersey Medical School, Rutgers, The State University of New Jersey, Newark, New Jersey. ^[5] Instituto de Fisiología, Facultad de Medicina, Universidad Austral de Chile, Valdivia, Chile. ^[6] Instituto de Inmunología, Facultad de Medicina, Universidad Austral de Chile, Valdivia, Chile; fabiolasanchez@uach.cl. ^[7]

关键词 endothelial function inflammation nitric oxide vasodilator-activated phosphoprotein