The critical role of SENP1-mediated GATA2 deSUMOylation in promoting endothelial activation in graft arteriosclerosis.

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作者： Cong,Qiu ^[1] ; Yuewen,Wang ^[2] ; Haige,Zhao ^[1] ; Lingfeng,Qin ^[2] ; Yanna,Shi ^[3] ; Xiaolong,Zhu ^[4] ; Lin,Song ^[5] ; Xiaofei,Zhou ^[1] ; Jian,Chen ^[2] ; Hong,Zhou ^[1] ; Haifeng,Zhang ^[2] ; George,Tellides ^[1] ; Wang,Min ^[2] ; Luyang,Yu ^[1]

作者单位： College of Life Sciences, Institute of Genetics and Regenerative Biology, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[1] Research Center for Air Pollution and Health, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[2] Department of Cardiothoracic Surgery, First Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[3] Interdepartmental Program in Vascular Biology and Therapeutics, Yale University School of Medicine, New Haven, Connecticut 06520, USA. ^[4] Department of Pathology and Surgery, Yale University School of Medicine, New Haven, Connecticut 06520, USA. ^[5] The First Affiliated Hospital, Center for Translational Medicine, Sun Yat-sen University, Guangzhou 510080, China. ^[6]

主题词动物(Animals);动脉硬化(Arteriosclerosis);半胱氨酸内肽酶类(Cysteine Endopeptidases);DNA(DNA);疾病恶化(Disease Progression);内肽酶类(Endopeptidases);内皮细胞(Endothelial Cells);内皮, 血管(Endothelium, Vascular);GATA2转录因子(GATA2 Transcription Factor);人类(Humans);炎症介导素类(Inflammation Mediators);白细胞(Leukocytes);男(雄)性(Male);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);模型, 生物学(Models, Biological);蛋白质结合(Protein Binding);蛋白质稳定性(Protein Stability);SUMO化(Sumoylation)

DOI 10.1038/ncomms15426

PMID 28569748

发布时间 2022-04-10

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Nature communications

2017年8卷

15426页

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The critical role of SENP1-mediated GATA2 deSUMOylation in promoting endothelial activation in graft arteriosclerosis.

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The critical role of SENP1-mediated GATA2 deSUMOylation in promoting endothelial activation in graft arteriosclerosis.

Nature communications

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Nature communications

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