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FGF21 does not require adipocyte AMP-activated protein kinase (AMPK) or the phosphorylation of acetyl-CoA carboxylase (ACC) to mediate improvements in whole-body glucose homeostasis.

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第一作者： Emilio P,Mottillo

第一单位： Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, 1280 Main St. W., Hamilton, Ontario, L8N 3Z5, Canada.

作者： Emilio P,Mottillo ^[1] ; Eric M,Desjardins ^[1] ; Andreas M,Fritzen ^[2] ; Vito Z,Zou ^[1] ; Justin D,Crane ^[1] ; Julian M,Yabut ^[1] ; Bente,Kiens ^[2] ; Derek M,Erion ^[3] ; Adhiraj,Lanba ^[4] ; James G,Granneman ^[5] ; Saswata,Talukdar ^[6] ; Gregory R,Steinberg ^[7]

作者单位： Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, 1280 Main St. W., Hamilton, Ontario, L8N 3Z5, Canada. ^[1] Section of Molecular Physiology, Department of Nutrition, Exercise and Sports, Faculty of Science, University of Copenhagen, Denmark. ^[2] Liver Disease Research, Takeda Pharmaceuticals, 35 Landsdowne Street, Cambridge, MA, 02139, USA. ^[3] Cardiovascular & Metabolic Diseases, Novartis Institute of Biomedical Research, 250 Massachusetts Avenue, Cambridge, MA, 02139, USA. ^[4] Wayne State University School of Medicine, Detroit, MI, 48201, USA. ^[5] Cardiometabolic Diseases, Merck Research Laboratories South San Francisco LLC, 630 Gateway Boulevard, South San Francisco, CA, 94080, USA. ^[6] Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, 1280 Main St. W., Hamilton, Ontario, L8N 3Z5, Canada.;Department of Biochemistry and Biomedical Sciences, McMaster University, 1280 Main St. W., Hamilton, Ontario, L8N 3Z5, Canada. ^[7]