Ubiquitin C-terminal hydrolase-L3 promotes interferon antiviral activity by stabilizing type I-interferon receptor.
第一作者:
Peng,Zhao
第一单位:
Institute of Infection and Immunity, Institutes of Biology and Medical Sciences, Soochow University, Suzhou, China; Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China.
作者:
主题词
抗病毒药(Antiviral Agents);半胱氨酸内肽酶类(Cysteine Endopeptidases);人类(Humans);免疫, 先天(Immunity, Innate);干扰素Ⅰ型(Interferon Type I);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);肽水解酶类(Peptide Hydrolases);受体, 干扰素αβ(Receptor, Interferon alpha-beta);泛素硫酯酶(Ubiquitin Thiolesterase)
DOI
10.1016/j.antiviral.2017.06.002
PMID
28583475
发布时间
2020-12-09
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Antiviral research
120-129页
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