Ubiquitin C-terminal hydrolase-L3 promotes interferon antiviral activity by stabilizing type I-interferon receptor.

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第一作者： Peng,Zhao

第一单位： Institute of Infection and Immunity, Institutes of Biology and Medical Sciences, Soochow University, Suzhou, China; Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China.

作者： Peng,Zhao ^[1] ; Tingting,Guo ^[1] ; Liping,Qian ^[1] ; Xiaofang,Wang ^[1] ; Yukang,Yuan ^[1] ; Qiao,Cheng ^[1] ; Yibo,Zuo ^[1] ; Jin,Liu ^[1] ; Ying,Miao ^[1] ; Qian,Feng ^[1] ; Liting,Zhang ^[1] ; Shuhua,Wu ^[2] ; Hui,Zheng ^[3]

作者单位： Institute of Infection and Immunity, Institutes of Biology and Medical Sciences, Soochow University, Suzhou, China; Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China. ^[1] Department of Emergency and Critical Care, The Second Affiliated Hospital of Soochow University, Suzhou, China. Electronic address: wushuhuasz@163.com. ^[2] Institute of Infection and Immunity, Institutes of Biology and Medical Sciences, Soochow University, Suzhou, China; Jiangsu Key Laboratory of Infection and Immunity, Soochow University, Suzhou, China. Electronic address: huizheng@suda.edu.cn. ^[3]

关键词 Antiviral response Deubiquitination Interferon SCF(β?TrCP)UCHL3