Knockdown of Mtfp1 can minimize doxorubicin cardiotoxicity by inhibiting Dnm1l-mediated mitochondrial fission.

导出 OA

翻译打印收藏纠错

作者： Lynn H H,Aung ^[1] ; Ruibei,Li ^[2] ; Bellur S,Prabhakar ^[1] ; Peifeng,Li ^[1]

作者单位： Department of Microbiology and Immunology, College of Medicine, University of Illinois at Chicago, Chicago, IL, USA. ^[1] School of Professional Studies, Northwestern University, Chicago, IL, USA. ^[2]

关键词 cardiotoxicity doxorubicin dyanmic-1-like (Dnm1l)mitochondrial fission mitochondrial fission process 1 (Mtfp1)

主题词动物(Animals);抗生素类, 抗肿瘤(Antibiotics, Antineoplastic);细胞系, 肿瘤(Cell Line, Tumor);DNA碎片裂(DNA Fragmentation);多柔比星(Doxorubicin);动力蛋白质类(Dynamins);基因表达调控(Gene Expression Regulation);遗传载体(Genetic Vectors);慢病毒属(Lentivirus);膜蛋白质类(Membrane Proteins);小鼠(Mice);线粒体(Mitochondria);肌细胞, 心脏(Myocytes, Cardiac);RNA, 小分子干扰(RNA, Small Interfering);信号传导(Signal Transduction)

DOI 10.1111/jcmm.13250

PMID 28643438

发布时间 2018-11-13

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览1

Journal of cellular and molecular medicine

2017年21卷12期

3394-3404页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Knockdown of Mtfp1 can minimize doxorubicin cardiotoxicity by inhibiting Dnm1l-mediated mitochondrial fission.

Journal of cellular and molecular medicine

相似文献

Journal of cellular and molecular medicine

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Knockdown of Mtfp1 can minimize doxorubicin cardiotoxicity by inhibiting Dnm1l-mediated mitochondrial fission.

Journal of cellular and molecular medicine

相似文献

Journal of cellular and molecular medicine

相关期刊

检索历史清除