An oncogenic axis of STAT-mediated BATF3 upregulation causing MYC activity in classical Hodgkin lymphoma and anaplastic large cell lymphoma.
第一作者:
A,Lollies
第一单位:
Institute of Cell Biology (Cancer Research), Faculty of Medicine, University of Duisburg-Essen, Essen, Germany.
作者:
医学主题词
碱性亮氨酸拉链转录因子类(Basic-Leucine Zipper Transcription Factors);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);细胞存活(Cell Survival);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);霍奇金病(Hodgkin Disease);人类(Humans);Janus激酶2(Janus Kinase 2);淋巴瘤, 大B细胞, 弥漫性(Lymphoma, Large B-Cell, Diffuse);淋巴瘤, 大细胞, 间变性(Lymphoma, Large-Cell, Anaplastic);癌基因(Oncogenes);启动区, 遗传(Promoter Regions, Genetic);原癌基因蛋白质c-myc(Proto-Oncogene Proteins c-myc);RNA, 小分子干扰(RNA, Small Interfering);STAT转录因子类(STAT Transcription Factors);信号传导(Signal Transduction);转录因子AP-1(Transcription Factor AP-1);转录激活(Transcriptional Activation);增量调节(Up-Regulation)
DOI
10.1038/leu.2017.203
PMID
28659618
发布时间
2018-12-11
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Leukemia
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