Exploiting Radiation-Induced Signaling to Increase the Susceptibility of Resistant Cancer Cells to Targeted Drugs: AKT and mTOR Inhibitors as an Example.

导出 OA

翻译打印收藏纠错

第一作者： Iris,Eke

第一单位： Radiation Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland. iris.eke@nih.gov.

作者： Iris,Eke ^[1] ; Adeola Y,Makinde ^[2] ; Molykutty J,Aryankalayil ^[2] ; Veit,Sandfort ^[3] ; Sanjeewani T,Palayoor ^[2] ; Barbara H,Rath ^[2] ; Lance,Liotta ^[4] ; Mariaelena,Pierobon ^[4] ; Emanuel F,Petricoin ^[4] ; Matthew F,Brown ^[2] ; Jayne M,Stommel ^[2] ; Mansoor M,Ahmed ^[5] ; C Norman,Coleman ^[6]

作者单位： Radiation Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland. iris.eke@nih.gov. ^[1] Radiation Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland. ^[2] Department of Radiology and Imaging Sciences, Clinical Center, National Institutes of Health, Bethesda, Maryland. ^[3] Center for Applied Proteomics and Molecular Medicine, George Mason University, Manassas, Virginia. ^[4] Radiation Research Program, National Cancer Institute, National Institutes of Health, Rockville, Maryland. ^[5] Radiation Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland.;Radiation Research Program, National Cancer Institute, National Institutes of Health, Rockville, Maryland. ^[6]