Pseudo-acetylation of multiple sites on human Tau proteins alters Tau phosphorylation and microtubule binding, and ameliorates amyloid beta toxicity.
第一作者:
Marianna Karina,Gorsky
第一单位:
Max Planck Institute for Biology of Ageing, Joseph-Stelzmann-Strasse 9b, 50931 Cologne, Germany; CECAD Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases, 50931, Cologne, Germany.
作者:
主题词
乙酰化作用(Acetylation);淀粉样β肽类(Amyloid beta-Peptides);动物(Animals);果蝇属(Drosophila);基因表达(Gene Expression);基因敲入技术(Gene Knock-In Techniques);基因敲除技术(Gene Knockout Techniques);人类(Humans);微管(Microtubules);模型, 动物(Models, Animal);磷酰化(Phosphorylation);蛋白质结合(Protein Binding);蛋白质多聚化(Protein Multimerization);蛋白质加工, 转译后(Protein Processing, Post-Translational);重组蛋白质类(Recombinant Proteins);tau蛋白质类(tau Proteins)
DOI
10.1038/s41598-017-10225-0
PMID
28855586
发布时间
2019-04-22
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