Syndecan-2 Attenuates Radiation-induced Pulmonary Fibrosis and Inhibits Fibroblast Activation by Regulating PI3K/Akt/ROCK Pathway via CD148.
第一作者:
Konstantin,Tsoyi
第一单位:
1 Division of Pulmonary and Critical Care Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts; and.
作者:
医学主题词
动物(Animals);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);女(雌)性(Female);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);肌成纤维细胞(Myofibroblasts);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);肺纤维化(Pulmonary Fibrosis);辐射损伤(Radiation Injuries);受体样蛋白质酪氨酸磷酸酶类, 3类(Receptor-Like Protein Tyrosine Phosphatases, Class 3);多配体蛋白聚糖2(Syndecan-2);胸部(Thorax);转化生长因子β(Transforming Growth Factor beta);rho相关激酶类(rho-Associated Kinases)
DOI
10.1165/rcmb.2017-0088OC
PMID
28886261
发布时间
2020-03-13
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