Neurotransmitter signalling via NMDA receptors leads to decreased T helper type 1-like and enhanced T helper type 2-like immune balance in humans.

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作者： Kanami,Orihara ^[1] ; Solomon O,Odemuyiwa ^[2] ; William P,Stefura ^[1] ; Ramses,Ilarraza ^[1] ; Kent T,HayGlass ^[2] ; Redwan,Moqbel ^[1]

作者单位： Department of Immunology, University of Manitoba, Winnipeg, MB, Canada. ^[1] Division of Pulmonary Medicine, Department of Medicine, University of Alberta, Edmonton, AB, Canada. ^[2]

关键词 NMDA CD4+ T cells NMDA-R Th1, Th2 glutamate glutamic acid human immune regulation

主题词 CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);细胞死亡(Cell Death);细胞系(Cell Line);细胞增殖(Cell Proliferation);细胞因子类(Cytokines);人类(Humans);白细胞介素10(Interleukin-10);淋巴细胞活化(Lymphocyte Activation);神经递质药(Neurotransmitter Agents);受体, 抗原, T细胞(Receptors, Antigen, T-Cell);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);T淋巴细胞, 辅助诱导(T-Lymphocytes, Helper-Inducer);Th1细胞(Th1 Cells);Th2细胞(Th2 Cells);增量调节(Up-Regulation)

DOI 10.1111/imm.12846

PMID 28940416

发布时间 2019-03-01

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Immunology

2018年153卷3期

368-379页

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Neurotransmitter signalling via NMDA receptors leads to decreased T helper type 1-like and enhanced T helper type 2-like immune balance in humans.

Immunology

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Immunology

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Neurotransmitter signalling via NMDA receptors leads to decreased T helper type 1-like and enhanced T helper type 2-like immune balance in humans.

Immunology

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Immunology

相关期刊

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