Mechanisms linked to differences in the mutagenic potential of 1,3-dinitropyrene and 1,8-dinitropyrene.
作者:
关键词
1,3-DNP, 1,3-dinitropyrene1,3-Dinitropyrene1,8-DNP, 1,8-dinitropyrene1,8-Dinitropyrene1-NP, 1-nitropyrene3-NBA, 3-nitrobenzanthroneAhR, aromatic hydrocarbon receptorApoptosisB[a]P, benzo[a]pyreneCM-H2DCFDA or H2DCFDA, 5-(and 6-)chloromethyl-2,7-dichlorodihydrofluorescein diacetateCYP, cytochrome P450Chk, checkpoint kinasesDDR, DNA damage responseDHE, dihydroethidiumDMSO, dimethyl sulfoxideDNA damageER, endoplasmic reticulumHoechst 33258, 2(2-(4-hydroxyphenyl)-6-benzimidazole-6-(1-methyl-4-piperazyl)benzimidazole hydrochloride)Hoechst 33342, 2′-(4-ethoxyphenyl)-2′,5′-bis-1H-benzimidazole hydrochloride)NR, nitro-reductasesnitro-PAHnitro substituted-polycyclic aromatic hydrocarbonNitro-PAHsPAH, polycyclic aromatic hydrocarbonPARP, poly(ADP-ribose) polymerasePFT, pifithrinPI, propidium iodidePM, particular matterRNS, reactive nitrogen speciesROS, reactive oxygen speciesSSB, single strand breaksUPR, unfolded protein responsefpg, formamidopyrimidine-DNA glycosylasezVAD-FMK, benzyolcarbonayl-Val-Ala-Asp-fluoromethyl ketoneγH2AX, phosphorylated H2AX
DOI
10.1016/j.toxrep.2014.07.009
PMID
28962260
发布时间
2020-09-30
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Toxicology reports
2014年1卷
459-473页
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