换一批
换一批Increased galectin-3 levels are associated with abdominal aortic aneurysm progression and inhibition of galectin-3 decreases elastase-induced AAA development.
第一单位：
Vascular Research Lab, FIIS-Fundación Jiménez Díaz-Autonoma University, Madrid, Spain.
作者：
主题词
动物(Animals);主动脉, 腹(Aorta, Abdominal);主动脉瘤, 腹(Aortic Aneurysm, Abdominal);血蛋白质类(Blood Proteins);病例对照研究(Case-Control Studies);细胞, 培养的(Cells, Cultured);趋化因子CCL5(Chemokine CCL5);扩张, 病理性(Dilatation, Pathologic);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);半乳糖凝集素3(Galectin 3);半乳糖凝集素类(Galectins);人类(Humans);小鼠, 近交C57BL(Mice, Inbred C57BL);肌, 平滑, 血管(Muscle, Smooth, Vascular);肌细胞, 平滑肌(Myocytes, Smooth Muscle);胰弹性蛋白酶(Pancreatic Elastase);果胶类(Pectins);磷酰化(Phosphorylation);RNA, 信使(RNA, Messenger);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction);时间因素(Time Factors);增量调节(Up-Regulation)
DOI
10.1042/CS20171142
PMID
28982723
发布时间
2021-12-04
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Clinical science (London, England
2707-2719页
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