Analysis of Nkx3.1:Cre-driven Erk5 deletion reveals a profound spinal deformity which is linked to increased osteoclast activity.
作者:
主题词
动物(Animals);骨质吸收(Bone Resorption);组织蛋白酶K(Cathepsin K);细胞计数(Cell Count);女(雌)性(Female);基因缺失(Gene Deletion);同源盒结构域蛋白质类(Homeodomain Proteins);整合酶类(Integrases);淋巴细胞活化(Lymphocyte Activation);男(雄)性(Male);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);丝裂原活化蛋白激酶7(Mitogen-Activated Protein Kinase 7);NFATC转录因子类(NFATC Transcription Factors);破骨细胞(Osteoclasts);骨生成(Osteogenesis);核因子κB受体活化因子(Receptor Activator of Nuclear Factor-kappa B);脊柱(Spine);转录因子(Transcription Factors)
DOI
10.1038/s41598-017-13346-8
PMID
29038439
发布时间
2024-02-10
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Scientific reports
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