Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.

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作者： Romel D,Mackelprang ^[1] ; Michael J,Bamshad ^[2] ; Jessica X,Chong ^[3] ; Xuanlin,Hou ^[2] ; Kati J,Buckingham ^[1] ; Kathryn,Shively ^[2] ; Guy,deBruyn ^[2] ; Nelly R,Mugo ^[4] ; James I,Mullins ^[1] ; M Juliana,McElrath ^[5] ; Jared M,Baeten ^[1] ; Connie,Celum ^[6] ; Mary J,Emond ^[7] ; Jairam R,Lingappa ^[7] ; Partners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams ^[8]

作者单位： Department of Global Health, University of Washington, Seattle, United States of America. ^[1] Department of Pediatrics, University of Washington, Seattle, United States of America. ^[2] Department of Genome Sciences, University of Washington, Seattle, United States of America. ^[3] Perinatal HIV Research Unit, University of Witwatersrand, Johannesburg, South Africa. ^[4] Partners in Health Research and Development, Kenya Medical Research Institute, Thika, Kenya. ^[5] Department of Microbiology, University of Washington, Seattle, United States of America. ^[6] Department of Medicine, University of Washington, Seattle, United States of America. ^[7] Vaccine and Infectious Disease Division, Fred Hutchinson Cancer Research Center, Seattle, United States of America. ^[8] Department of Epidemiology, University of Washington, Seattle, United States of America. ^[9] Department of Biostatistics, University of Washington, Seattle, United States of America. ^[10]