Mechanisms of RhoA inactivation and CDC42 and Rac1 activation during zebrafish optic nerve regeneration.

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作者： Toru,Matsukawa ^[1] ; Kazune,Morita ^[2] ; Shou,Omizu ^[2] ; Satoru,Kato ^[3] ; Yoshiki,Koriyama ^[4]

作者单位： Faculty of Science and Engineering, Department of Life Science, Setsunan University, Neyagawa, Osaka, 572-8508, Japan. Electronic address: matsukawa@lif.setsunan.ac.jp. ^[1] Faculty of Science and Engineering, Department of Life Science, Setsunan University, Neyagawa, Osaka, 572-8508, Japan. ^[2] Wellness Promotion Science Center, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, 920-0942, Japan. ^[3] Graduate School and Faculty of Pharmaceutical Sciences, Suzuka University of Medical Science, Suzuka, 513-8670, Japan. ^[4]

关键词 CDC42 Rhotekin TAX1BP3 Wnt10a Wnt5b β-Catenin

主题词动物(Animals);酶激活(Enzyme Activation);酶抑制剂(Enzyme Inhibitors);神经再生(Nerve Regeneration);视神经(Optic Nerve);斑马鱼(Zebrafish);cdc42 GTP结合蛋白质(cdc42 GTP-Binding Protein);rac1 GTP结合蛋白质(rac1 GTP-Binding Protein);ρA GTP结合蛋白质(rhoA GTP-Binding Protein)

DOI 10.1016/j.neuint.2017.11.004

PMID 29129556

发布时间 2019-05-28

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Neurochemistry international

2018年112卷

71-80页

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Mechanisms of RhoA inactivation and CDC42 and Rac1 activation during zebrafish optic nerve regeneration.

Neurochemistry international

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Neurochemistry international

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Mechanisms of RhoA inactivation and CDC42 and Rac1 activation during zebrafish optic nerve regeneration.

Neurochemistry international

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Neurochemistry international

相关期刊

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