首页> Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism> Induction of the cell survival kinase Sgk1: A possible novel mechanism for α-phenyl-N-tert-butyl nitrone in experimental stroke.

Induction of the cell survival kinase Sgk1: A possible novel mechanism for α-phenyl-N-tert-butyl nitrone in experimental stroke.

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第一作者： Catherine,McCaig

第一单位： 1 Molecular and Clinical Sciences Research Institute, St Georges University of London, London, UK.

作者： Catherine,McCaig ^[1] ; Paris,Ataliotis ^[2] ; Anan,Shtaya ^[1] ; Ayan S,Omar ^[1] ; A Richard,Green ^[3] ; Clive N,Kind ^[4] ; Anthony C,Pereira ^[5] ; Aniko,Naray-Fejes-Toth ^[6] ; Geza,Fejes-Toth ^[6] ; Rafael J,Yáñez-Muñoz ^[7] ; James T,Murray ^[8] ; Atticus H,Hainsworth ^[5]

作者单位： 1 Molecular and Clinical Sciences Research Institute, St Georges University of London, London, UK. ^[1] 2 Institute for Medical & Biomedical Education, St George's University of London, London, UK. ^[2] 3 School of Life Sciences, University of Nottingham, Nottingham, UK. ^[3] 4 Leicester School of Pharmacy, De Montfort University, Leicester, UK. ^[4] 1 Molecular and Clinical Sciences Research Institute, St Georges University of London, London, UK.;5 Department of Neurology, St George's University Hospitals NHS Foundation Trust, London, UK. ^[5] 6 Molecular & Systems Biology Department, Geisel School of Medicine at Dartmouth, Lebanon, NH, USA. ^[6] 7 AGCTlab.org, Centre for Biomedical Sciences, School of Biological Sciences, Royal Holloway, University of London, Egham, Surrey, UK. ^[7] 8 School of Biochemistry and Immunology, Trinity Biomedical Sciences Institute, Trinity College Dublin, Dublin 2, Ireland. ^[8]

关键词 Acute stroke animal models focal ischemia lacunar infarcts neuroprotection