Cocaine- and amphetamine-regulated transcript peptide in the nucleus accumbens shell inhibits cocaine-induced locomotor sensitization to transient over-expression of α-Ca<sup>2+</sup> /calmodulin-dependent protein kinase II.
第一作者:
Lixia,Xiong
第一单位:
Department of Pathophysiology, College of Basic Medicine, Nanchang University, Nanchang, Jiangxi, China.
作者:
医学主题词
动物(Animals);动物, 新生(Animals, Newborn);钙(Calcium);钙-钙调素依赖性蛋白激酶2型(Calcium-Calmodulin-Dependent Protein Kinase Type 2);可卡因(Cocaine);多巴胺摄取抑制剂(Dopamine Uptake Inhibitors);剂量效应关系, 药物(Dose-Response Relationship, Drug);基因表达调控(Gene Expression Regulation);免疫沉淀法(Immunoprecipitation);移动(Locomotion);男(雄)性(Male);神经组织蛋白质类(Nerve Tissue Proteins);神经元(Neurons);伏核(Nucleus Accumbens);肽碎片(Peptide Fragments);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 多巴胺D3(Receptors, Dopamine D3);转染(Transfection)
DOI
10.1111/jnc.14289
PMID
29313985
发布时间
2025-01-03
- 浏览6
Journal of neurochemistry
289-303页
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