MK2 puts an additional brake on RIPK1 cytotoxic potential.

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作者： Yves,Dondelinger ^[1] ; Tom,Delanghe ^[2] ; Mathieu M J,Bertrand ^[1]

作者单位： VIB Center for Inflammation Research, Technologiepark 927, 9052, Zwijnaarde-Ghent, Belgium. ^[1] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, 9052, Zwijnaarde-Ghent, Belgium. ^[2] VIB Center for Inflammation Research, Technologiepark 927, 9052, Zwijnaarde-Ghent, Belgium. mathieu.bertrand@irc.vib-ugent.be. ^[3] Department of Biomedical Molecular Biology, Ghent University, Technologiepark 927, 9052, Zwijnaarde-Ghent, Belgium. mathieu.bertrand@irc.vib-ugent.be. ^[4]

主题词细胞死亡(Cell Death);人类(Humans);炎症(Inflammation);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);磷酰化(Phosphorylation);受体作用蛋白丝氨酸苏氨酸激酶类(Receptor-Interacting Protein Serine-Threonine Kinases)

DOI 10.1038/s41418-017-0009-z

PMID 29317761

发布时间 2021-12-04

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准确一般字义偏差语意偏离译文作用小提交