Bridging pro-inflammatory signals, synaptic transmission and protection in spinal explants in vitro.
第一作者:
M,Medelin
第一单位:
Department of Life Sciences, University of Trieste, 34127, Trieste, Italy.
作者:
主题词
动物(Animals);炎症趋化因子类(Chemokines);女(雌)性(Female);神经胶质增生(Gliosis);植入物, 实验性(Implants, Experimental);炎症(Inflammation);炎症介导素类(Inflammation Mediators);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);小鼠, 近交C57BL(Mice, Inbred C57BL);神经生长因子(Nerve Growth Factor);器官培养技术(Organ Culture Techniques);受体, trkA(Receptor, trkA);受体, trkB(Receptor, trkB);受体, GABA(Receptors, GABA);信号传导(Signal Transduction);脊髓(Spinal Cord);应激, 生理学(Stress, Physiological);突触(Synapses);突触传递(Synaptic Transmission);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1186/s13041-018-0347-x
PMID
29334986
发布时间
2018-11-13
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