Lactobacillus acidophilus Improves Intestinal Inflammation in an Acute Colitis Mouse Model by Regulation of Th17 and Treg Cell Balance and Fibrosis Development.
作者:
主题词
肌动蛋白类(Actins);动物(Animals);细胞, 培养的(Cells, Cultured);结肠炎(Colitis);胶原Ⅰ型(Collagen Type I);结肠(Colon);细胞因子类(Cytokines);纤维化(Fibrosis);基因表达调控(Gene Expression Regulation);白细胞介素10(Interleukin-10);肠黏膜(Intestinal Mucosa);嗜酸乳杆菌(Lactobacillus acidophilus);男(雄)性(Male);小鼠, 近交C57BL(Mice, Inbred C57BL);肌成纤维细胞(Myofibroblasts);有益菌种(Probiotics);随机分配(Random Allocation);无特异性病原体有机物(Specific Pathogen-Free Organisms);脾(Spleen);T淋巴细胞, 调节性(T-Lymphocytes, Regulatory);Th17细胞(Th17 Cells)
DOI
10.1089/jmf.2017.3990
PMID
29336663
发布时间
2018-12-02
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Journal of medicinal food
215-224页
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