Prolonged dipyridamole administration reduces myocardial perfusion defects in experimental chronic Chagas cardiomyopathy.

导出原文传递

翻译打印收藏纠错

作者： Denise Mayumi,Tanaka ^[1] ; Luciano Fonseca Lemos,de Oliveira ^[1] ; José Antônio,Marin-Neto ^[1] ; Minna Moreira Dias,Romano ^[1] ; Eduardo Elias Vieira,de Carvalho ^[2] ; Antonio Carlos Leite,de Barros Filho ^[1] ; Fernando Fonseca França,Ribeiro ^[1] ; Jorge Mejia,Cabeza ^[3] ; Carla Duque,Lopes ^[1] ; Camila Godoy,Fabricio ^[1] ; Norival,Kesper ^[4] ; Henrique Turin,Moreira ^[1] ; Lauro,Wichert-Ana ^[1] ; André,Schmidt ^[1] ; Maria de Lourdes,Higuchi ^[5] ; Edécio,Cunha-Neto ^[5] ; Marcus Vinícius,Simões ^[6]

作者单位： Medical School of Ribeirao Preto, University of São Paulo, Sao Paulo, Brazil. ^[1] Department of Applied Physical Therapy, Institute of Health Sciences, Federal University of Triangulo Mineiro, Minas Gerais, Brazil. ^[2] Hospital Israelita Albert Einstein, Sao Paulo, Brazil. ^[3] Instituto de Medicina Tropical, Faculty of Medicine, University os Sao Paulo, Sao Paulo, Brazil. ^[4] Heart Institute (InCor), Faculty of Medicine, University of Sao Paulo, Sao Paulo, Brazil. ^[5] Medical School of Ribeirao Preto, University of São Paulo, Sao Paulo, Brazil. msimoes@fmrp.usp.br. ^[6]

关键词 Chagas cardiomyopathy coronary microcirculation dipyridamole hamsters ventricular dysfunction