New thiazolidinedione LPSF/GQ-2 inhibits NFκB and MAPK activation in LPS-induced acute lung inflammation.
第一单位:
Laboratory of Ultrastructure, Aggeu Magalhães Institute (IAM), Recife, PE, Brazil; Postgraduate Program in Biosciences and Biotechnology for Health (PPGBBS), Oswaldo Cruz Foundation (FIOCRUZ-PE)/Aggeu Magalhães Institute (IAM), Recife, PE, Brazil.
作者:
主题词
急性肺损伤(Acute Lung Injury);动物(Animals);消炎药(Anti-Inflammatory Agents);细胞因子类(Cytokines);疾病模型, 动物(Disease Models, Animal);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);人类(Humans);炎症介导素类(Inflammation Mediators);脂多糖类(Lipopolysaccharides);男(雄)性(Male);小鼠(Mice);NF-κB(NF-kappa B);过氧化物酶体增殖物激活受体(Peroxisome Proliferator-Activated Receptors);肺炎(Pneumonia);信号传导(Signal Transduction);噻唑烷二酮类(Thiazolidinediones)
DOI
10.1016/j.intimp.2018.02.011
PMID
29475100
发布时间
2020-12-09
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