Overexpression of Tpl2 is linked to imatinib resistance and activation of MEK-ERK and NF-κB pathways in a model of chronic myeloid leukemia.
第一作者:
Anna,Chorzalska
第一单位:
Signal Transduction Lab, Division of Hematology/Oncology, Rhode Island Hospital, Warren Alpert Medical School, Brown University, Providence, RI, USA.
作者:
医学主题词
动物(Animals);细胞系, 肿瘤(Cell Line, Tumor);疾病模型, 动物(Disease Models, Animal);抗药性, 肿瘤(Drug Resistance, Neoplasm);融合蛋白质类, bcr-abl(Fusion Proteins, bcr-abl);人类(Humans);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);MAP激酶激酶激酶类(MAP Kinase Kinase Kinases);MAP激酶信号系统(MAP Kinase Signaling System);丝裂原激活蛋白激酶激酶类(Mitogen-Activated Protein Kinase Kinases);模型, 生物学(Models, Biological);NF-κB(NF-kappa B);磷肽类(Phosphopeptides);蛋白质组学(Proteomics);原癌基因蛋白质类(Proto-Oncogene Proteins);信号传导(Signal Transduction);src族激酶类(src-Family Kinases)
DOI
10.1002/1878-0261.12186
PMID
29485707
发布时间
2020-04-23
- 浏览8
Molecular oncology
630-647页
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