Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering.

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作者： Larry A,Cogburn ^[1] ; Danielle N,Smarsh ^[2] ; Xiaofei,Wang ^[3] ; Nares,Trakooljul ^[2] ; Wilfrid,Carré ^[4] ; Harold B,White ^[2]

作者单位： Department of Animal and Food Sciences, University of Delaware, Newark, DE, 19716, USA. cogburn@udel.edu. ^[1] Department of Animal and Food Sciences, University of Delaware, Newark, DE, 19716, USA. ^[2] Present Address: Department of Animal Science, The Pennsylvania State University, University Park, PA, 16802, USA. ^[3] Present Address: Department of Biological Sciences, Tennessee State University, Nashville, TN, 37209, USA. ^[4] Present Address: Leibniz Institute for Farm Animal Biology (FBN), Institute for Genome Biology, Wilhelm-Stahl-Allee 2, 18196, Dummerstorf, Germany. ^[5] Present Address: Laboratoire de Génétique Moléculaire et Génomique, CHU Pontchaillou, 35033, Rennes, France. ^[6] Department of Chemistry and Biochemistry, University of Delaware, Newark, DE, 19716, USA. ^[7]

关键词 Acetyl CoA deficiency Chicken flavoproteome Coagulation factors Expression profiling Feather keratin Impaired lipid metabolism Microarray analysis Protease inhibitors Protein catabolism Riboflavin deficiency Sudden embryonic death β-oxidation