High-density lipoprotein protects cardiomyocytes against necrosis induced by oxygen and glucose deprivation through SR-B1, PI3K, and AKT1 and 2.
第一作者:
Kristina K,Durham
第一单位:
Thrombosis and Atherosclerosis Research Institute, McMaster University and Hamilton Health Sciences, 237 Barton St. E. Hamilton, Ontario, Canada L8L 2X2.
作者:
主题词
动物(Animals);葡萄糖(Glucose);人类(Humans);脂蛋白类, HDL(Lipoproteins, HDL);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);肌细胞, 心脏(Myocytes, Cardiac);坏死(Necrosis);氧(Oxygen);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction)
DOI
10.1042/BCJ20170703
PMID
29523748
发布时间
2022-04-08
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The Biochemical journal
1253-1265页
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