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NF-κB inhibition by dimethylaminoparthenolide radiosensitizes non-small-cell lung carcinoma by blocking DNA double-strand break repair.

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第一作者： Peter V,Deraska

第一单位： 1Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA USA.

作者： Peter V,Deraska ^[1] ; Colin,O'Leary ^[1] ; Hunter D,Reavis ^[1] ; Shelby,Labe ^[1] ; Tru-Khang,Dinh ^[1] ; Jean-Bernard,Lazaro ^[2] ; Christopher,Sweeney ^[3] ; Alan D,D'Andrea ^[4] ; David,Kozono ^[1]

作者单位： 1Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA USA. ^[1] 1Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA USA.;2Center for DNA Damage and Repair, Dana-Farber Cancer Institute, Boston, MA USA. ^[2] 3Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA USA. ^[3] 1Department of Radiation Oncology, Dana-Farber Cancer Institute, Boston, MA USA.;2Center for DNA Damage and Repair, Dana-Farber Cancer Institute, Boston, MA USA.;4Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA USA. ^[4]

DOI 10.1038/s41420-017-0008-3

PMID 29531807

发布时间 2020-10-01

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NF-κB inhibition by dimethylaminoparthenolide radiosensitizes non-small-cell lung carcinoma by blocking DNA double-strand break repair.

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NF-κB inhibition by dimethylaminoparthenolide radiosensitizes non-small-cell lung carcinoma by blocking DNA double-strand break repair.

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