Dehydroepiandrosterone-induced activation of mTORC1 and inhibition of autophagy contribute to skeletal muscle insulin resistance in a mouse model of polycystic ovary syndrome.

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作者： Xi,Song ^[1] ; Qiyang,Shen ^[1] ; Liting,Fan ^[1] ; Qiuxiao,Yu ^[1] ; Xiao,Jia ^[2] ; Yu,Sun ^[3] ; Wenpei,Bai ^[3] ; Jihong,Kang ^[1]

作者单位： Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Peking University Health Science Center, Beijing, China. ^[1] Institute of Infectious Diseases, Beijing Ditan Hospital, Capital Medical University, Beijing, China. ^[2] Department of Obstetrics and Gynecology, Beijing Shijitan Hospital, Beijing, China. ^[3]

关键词 autophagy dehydroepiandrosterone (DHEA)mTOR polycystic ovary syndrome (PCOS)skeletal muscle insulin resistance

DOI 10.18632/oncotarget.24190

PMID 29552281

发布时间 2019-11-20

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