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ZIP4 Promotes Pancreatic Cancer Progression by Repressing ZO-1 and Claudin-1 through a ZEB1-Dependent Transcriptional Mechanism.

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作者单位: Department of Medicine, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma. [1] Department of Surgery, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma. [2] Department of Pathology, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma. [3] Department of Biostatistics and Epidemiology, College of Public Health, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma. [4] Department of Pathology, Peking Union Hospital, Peking Union Medical College, Beijing, China. [5] The Sidney Kimmel Comprehensive Cancer Center and Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, Maryland. [6] Department of Oncology, Mayo Clinic, Rochester, Minnesota. [7] Schulze Center for Novel Therapeutics, Mayo Clinic, Rochester, Minnesota. [8] Department of Integrative Biology and Pharmacology, the University of Texas Medical School at Houston, Houston, Texas. [9] Department of Pharmacology and Pediatrics, University of California at San Diego, La Jolla, California. [10] Department of Medicine, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma. Min-Li@ouhsc.edu. [11]
DOI 10.1158/1078-0432.CCR-18-0263
PMID 29615456
发布时间 2024-04-23
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Clinical cancer research : an official journal of the American Association for Cancer Research

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