A novel interaction between CX<sub>3</sub>CR<sub>1</sub> and CCR<sub>2</sub> signalling in monocytes constitutes an underlying mechanism for persistent vincristine-induced pain.
作者:
主题词
动物(Animals);抗肿瘤药, 植物(Antineoplastic Agents, Phytogenic);苯并恶嗪类(Benzoxazines);钙结合蛋白质类(Calcium-Binding Proteins);细胞系, 转化(Cell Line, Transformed);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);基因表达调控(Gene Expression Regulation);痛觉过敏(Hyperalgesia);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);微丝蛋白质类(Microfilament Proteins);单核细胞(Monocytes);疼痛(Pain);哌啶类(Piperidines);受体, CCR2(Receptors, CCR2);受体, CXCR3(Receptors, CXCR3);信号传导(Signal Transduction);长春新碱(Vincristine)
DOI
10.1186/s12974-018-1116-6
PMID
29625610
发布时间
2022-01-29
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