Impaired Ca<sup>2+</sup> cycling of nonischemic myocytes contributes to sarcomere dysfunction early after myocardial infarction.

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作者： Annette,Kronenbitter ^[1] ; Florian,Funk ^[1] ; Katarzyna,Hackert ^[1] ; Simone,Gorreßen ^[1] ; Dennis,Glaser ^[2] ; Peter,Boknik ^[2] ; Gereon,Poschmann ^[3] ; Kai,Stühler ^[3] ; Malgorzata,Isić ^[4] ; Martina,Krüger ^[4] ; Joachim P,Schmitt ^[5]

作者单位： Institute of Pharmacology and Clinical Pharmacology, University Hospital Düsseldorf, Germany; Cardiovascular Research Institute Düsseldorf (CARID), Heinrich-Heine-University, Düsseldorf, Germany. ^[1] Institute of Pharmacology and Toxicology, University Hospital Münster, Germany. ^[2] Molecular Proteomics Laboratory, Biological and Medical Research Center (BMFZ), Institute of Molecular Medicine, University Hospital Düsseldorf, Germany. ^[3] Institute of Cardiovascular Physiology, University of Düsseldorf, Germany; Cardiovascular Research Institute Düsseldorf (CARID), Heinrich-Heine-University, Düsseldorf, Germany. ^[4] Institute of Pharmacology and Clinical Pharmacology, University Hospital Düsseldorf, Germany; Cardiovascular Research Institute Düsseldorf (CARID), Heinrich-Heine-University, Düsseldorf, Germany. Electronic address: joachim.schmitt@uni-duesseldorf.de. ^[5]

关键词 Myocardial infarction Myocyte calcium cycling Phospholamban Remote myocardium SERCA2a Sarcomere function