换一批Low doses of LPS exacerbate the inflammatory response and trigger death on TLR3-primed human monocytes.
第一作者：
Marta,Monguió-Tortajada
第一单位：
REMAR-IVECAT Group, Health Science Research Institute Germans Trias i Pujol, Can Ruti Campus, Badalona, Spain.;Department of Cell Biology, Physiology and Immunology, Universitat Autònoma de Barcelona, Bellaterra, Spain.
作者：
医学主题词
细胞凋亡(Apoptosis);基因表达调控(Gene Expression Regulation);人类(Humans);咪唑类(Imidazoles);免疫, 先天(Immunity, Innate);白细胞介素6(Interleukin-6);脂多糖类(Lipopolysaccharides);单核细胞(Monocytes);聚肌胞苷酸(Poly I-C);受体, 肿瘤坏死因子, Ⅰ型(Receptors, Tumor Necrosis Factor, Type I);受体, 肿瘤坏死因子, Ⅱ型(Receptors, Tumor Necrosis Factor, Type II);信号传导(Signal Transduction);Toll样受体3(Toll-Like Receptor 3);Toll样受体4(Toll-Like Receptor 4);Toll样受体7(Toll-Like Receptor 7);Toll样受体8(Toll-Like Receptor 8);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1038/s41419-018-0520-2
PMID
29717111
发布时间
2024-03-26
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