Epigallocatechin-3-Gallate Regulates Anti-Inflammatory Action Through 67-kDa Laminin Receptor-Mediated Tollip Signaling Induction in Lipopolysaccharide-Stimulated Human Intestinal Epithelial Cells.

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第一作者： Eui-Baek,Byun

第一单位： Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Republic of Korea.

作者： Eui-Baek,Byun ^[1] ; Woo Sik,Kim ^[1] ; Nak-Yun,Sung ^[2] ; Eui-Hong,Byun ^[2]

作者单位： Advanced Radiation Technology Institute, Korea Atomic Energy Research Institute, Jeongeup, Republic of Korea. ^[1] Department of Food Science and Technology, Kongju National University, Yesan, Republic of Korea. ^[2]

关键词 67-kDa laminin receptor Epigallocatechin-3-gallate HT-29 cells IL-8 production Toll-like receptor 4 Tollip induction

主题词消炎药(Anti-Inflammatory Agents);儿茶素(Catechin);HT29细胞(HT29 Cells);人类(Humans);肠黏膜(Intestinal Mucosa);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);脂多糖类(Lipopolysaccharides);受体, 层黏连蛋白(Receptors, Laminin);信号传导(Signal Transduction)

DOI 10.1159/000489447

PMID 29723847

发布时间 2019-02-12

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Cellular physiology and biochemistry

2018年46卷5期

2072-2081页

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Epigallocatechin-3-Gallate Regulates Anti-Inflammatory Action Through 67-kDa Laminin Receptor-Mediated Tollip Signaling Induction in Lipopolysaccharide-Stimulated Human Intestinal Epithelial Cells.

Cellular physiology and biochemistry

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Epigallocatechin-3-Gallate Regulates Anti-Inflammatory Action Through 67-kDa Laminin Receptor-Mediated Tollip Signaling Induction in Lipopolysaccharide-Stimulated Human Intestinal Epithelial Cells.

Cellular physiology and biochemistry

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Cellular physiology and biochemistry

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