Actin Dysfunction Induces Cell Cycle Delay at G2/M with Sustained ERK and RSK Activation in IMR-90 Normal Human Fibroblasts.
第一作者:
Deepmala,Shrestha
第一单位:
Department of Biochemistry, College of Life Science and Biotechnology, Yonsei University, Seoul 03722, Korea.
作者:
主题词
肌动蛋白类(Actins);CDC2蛋白激酶(CDC2 Protein Kinase);细胞周期蛋白质类(Cell Cycle Proteins);细胞松弛素D(Cytochalasin D);酶激活(Enzyme Activation);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);成纤维细胞(Fibroblasts);人类(Humans);MAP激酶信号系统(MAP Kinase Signaling System);核蛋白质类(Nuclear Proteins);磷酰化(Phosphorylation);蛋白质加工, 转译后(Protein Processing, Post-Translational);蛋白酪氨酸激酶类(Protein-Tyrosine Kinases);核糖体蛋白质S6激酶类, 90-kDa(Ribosomal Protein S6 Kinases, 90-kDa);cdc25磷酸酶类(cdc25 Phosphatases)
DOI
10.14348/molcells.2018.2266
PMID
29754473
发布时间
2024-02-04
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Molecules and cells
436-443页
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