The TP53-Induced Glycolysis and Apoptosis Regulator mediates cooperation between HTLV-1 p30<sup>II</sup> and the retroviral oncoproteins Tax and HBZ and is highly expressed in an in vivo xenograft model of HTLV-1-induced lymphoma.

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作者： Tetiana,Hutchison ^[1] ; Aditi,Malu ^[1] ; Laçin,Yapindi ^[1] ; Rachel,Bergeson ^[1] ; Kendra,Peck ^[1] ; Megan,Romeo ^[1] ; Carolyn,Harrod ^[1] ; Jordan,Pope ^[1] ; Louisa,Smitherman ^[1] ; Wesleigh,Gwinn ^[1] ; Lee,Ratner ^[2] ; Courtney,Yates ^[3] ; Robert,Harrod ^[4]

作者单位： Laboratory of Molecular Virology, Department of Biological Sciences, and The Dedman College Center for Drug Discovery, Design & Delivery, Southern Methodist University, 6501 Airline Drive, 334-DLS, Dallas, TX 75275-0376, United States. ^[1] Departments of Medicine and Molecular Microbiology, Washington University School of Medicine, St. Louis, MO 63110, United States. ^[2] Laboratory Animal Resource Center, Southern Methodist University, Dallas, TX 75275, United States. ^[3] Laboratory of Molecular Virology, Department of Biological Sciences, and The Dedman College Center for Drug Discovery, Design & Delivery, Southern Methodist University, 6501 Airline Drive, 334-DLS, Dallas, TX 75275-0376, United States. Electronic address: rharrod@smu.edu. ^[4]

关键词 ATLL Angiogenesis HBZ HTLV-1 Lymphoma ROS TIGAR Tax Xenograft p30(II)