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Proteasomal degradation of the histone acetyl transferase p300 contributes to beta-cell injury in a diabetes environment.

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第一作者： Lucie,Ruiz

第一单位： IGF, CNRS, INSERM, University of Montpellier, Montpellier, France.

作者： Lucie,Ruiz ^[1] ; Tatyana,Gurlo ^[2] ; Magalie A,Ravier ^[1] ; Anne,Wojtusciszyn ^[3] ; Julia,Mathieu ^[1] ; Matthew R,Brown ^[4] ; Christophe,Broca ^[5] ; Gyslaine,Bertrand ^[1] ; Peter C,Butler ^[2] ; Aleksey V,Matveyenko ^[4] ; Stéphane,Dalle ^[1] ; Safia,Costes ^[6]

作者单位： IGF, CNRS, INSERM, University of Montpellier, Montpellier, France. ^[1] Larry L. Hillblom Islet Research Center, David Geffen School of Medicine, University of California Los Angeles, Los Angeles, CA, USA. ^[2] IGF, CNRS, INSERM, University of Montpellier, Montpellier, France.;Laboratory of Cell Therapy for Diabetes (LTCD), Institute for Regenerative Medicine and Biotherapy (IRMB), University Hospital of Montpellier, Montpellier, France.;Department of Endocrinology, Diabetes, and Nutrition, University Hospital of Montpellier, Montpellier, France. ^[3] Department of Physiology and Biomedical Engineering, Mayo Clinic School of Medicine, Mayo Clinic, Rochester, MN, USA. ^[4] Laboratory of Cell Therapy for Diabetes (LTCD), Institute for Regenerative Medicine and Biotherapy (IRMB), University Hospital of Montpellier, Montpellier, France. ^[5] IGF, CNRS, INSERM, University of Montpellier, Montpellier, France. safia.costes@igf.cnrs.fr. ^[6]