Combination of anti-citrullinated protein antibodies and rheumatoid factor is associated with increased systemic inflammatory mediators and more rapid progression from preclinical to clinical rheumatoid arthritis.

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作者： Nithya,Lingampalli ^[1] ; Jeremy,Sokolove ^[1] ; Lauren J,Lahey ^[1] ; Jess D,Edison ^[2] ; William R,Gilliland ^[3] ; V Michael,Holers ^[4] ; Kevin D,Deane ^[5] ; William H,Robinson ^[6]

作者单位： VA Palo Alto Health Care System, 3801 Miranda Ave, C4-191, Palo Alto, CA 94304, United States; Division of Immunology and Rheumatology, Stanford University, 269 Campus Drive, CCSR 4135, Stanford, CA 94305, United States. ^[1] Rheumatology Service, Department of Medicine, Walter Reed National Military Medical Center, 8901 Wisconsin Avenue, Bethesda, MD 20889, United States. ^[2] Uniformed Services University of the Health Sciences, 4301 Jones Bridge Road, Bethesda, MD 20814, United States. ^[3] Division of Rheumatology, University of Colorado Denver School of Medicine, 1775 Aurora Court, Aurora, CO 80045, United States. ^[4] Division of Rheumatology, University of Colorado Denver School of Medicine, 1775 Aurora Court, Aurora, CO 80045, United States. Electronic address: kevin.deane@ucdenver.edu. ^[5] VA Palo Alto Health Care System, 3801 Miranda Ave, C4-191, Palo Alto, CA 94304, United States; Division of Immunology and Rheumatology, Stanford University, 269 Campus Drive, CCSR 4135, Stanford, CA 94305, United States. Electronic address: w.robinson@stanford.edu. ^[6]

关键词 Anti-citrullinated protein antibodies (ACPA)Cytokines Rheumatoid arthritis Rheumatoid factor (RF)