Dicalcium Silicate Induced Proinflammatory Responses through TLR2-Mediated NF-<i>κ</i>B and JNK Pathways in the Murine RAW 264.7 Macrophage Cell Line.

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作者： Shixiang,Lai ^[1] ; Liangjiao,Chen ^[1] ; Wei,Cao ^[1] ; Shiman,Cui ^[1] ; Xingyang,Li ^[1] ; Wenchao,Zhong ^[1] ; Mingyu,Ma ^[2] ; Qingbin,Zhang ^[1]

作者单位： Key Laboratory of Oral Medicine, Guangzhou Institute of Oral Disease, Stomatology Hospital of Guangzhou Medical University, Guangzhou 510140, China. ^[1] The Second Affiliated Hospital of Guangzhou Medical University, Guangzhou 510260, China. ^[2]

主题词动物(Animals);细胞凋亡(Apoptosis);钙化合物(Calcium Compounds);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);小鼠(Mice);显微镜检查, 电子, 扫描(Microscopy, Electron, Scanning);NF-κB(NF-kappa B);硅酸盐类(Silicates);TNF受体相关死亡结构域蛋白质(TNF Receptor-Associated Death Domain Protein);TNF受体相关因子2(TNF Receptor-Associated Factor 2);Toll样受体2(Toll-Like Receptor 2);X线衍射(X-Ray Diffraction)

DOI 10.1155/2018/8167932

PMID 29853794

发布时间 2019-01-24

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Mediators of inflammation

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Dicalcium Silicate Induced Proinflammatory Responses through TLR2-Mediated NF-<i>κ</i>B and JNK Pathways in the Murine RAW 264.7 Macrophage Cell Line.

Mediators of inflammation

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Mediators of inflammation

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Dicalcium Silicate Induced Proinflammatory Responses through TLR2-Mediated NF-<i>κ</i>B and JNK Pathways in the Murine RAW 264.7 Macrophage Cell Line.

Mediators of inflammation

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Mediators of inflammation

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