Mitochondrial Dysfunctions Regulated Radioresistance through Mitochondria-to-Nucleus Retrograde Signaling Pathway of NF-κB/PI3K/AKT2/mTOR.
第一作者:
Yuehua,Wei
第一单位:
a Department of Radiation and Medical Oncology.;b Hubei Key Laboratory of Tumor Biological Behaviors.;d Cancer Center, Renmin Hospital of Wuhan University, Wuhan, 430060, China.
作者:
医学主题词
细胞凋亡(Apoptosis);细胞周期(Cell Cycle);细胞系, 肿瘤(Cell Line, Tumor);细胞核(Cell Nucleus);DNA损伤(DNA Damage);DNA修复(DNA Repair);DNA, 线粒体(DNA, Mitochondrial);人类(Humans);线粒体(Mitochondria);NF-κB(NF-kappa B);氧化性应激(Oxidative Stress);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);辐射耐受性(Radiation Tolerance);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases)
DOI
10.1667/RR15021.1
PMID
29863983
发布时间
2021-12-04
- 浏览15
Radiation research
204-215页
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