Carbon monoxide attenuates lipopolysaccharide-induced lung injury by mitofusin proteins via p38 MAPK pathway.
第一作者:
Shu-An,Dong
第一单位:
Department of Anesthesiology, Tianjin Nankai Hospital, Tianjin Medical University, Tianjin, China.
作者:
医学主题词
急性肺损伤(Acute Lung Injury);动物(Animals);疾病模型, 动物(Disease Models, Animal);药物评价, 临床前(Drug Evaluation, Preclinical);GTP磷酸水解酶类(GTP Phosphohydrolases);人类(Humans);咪唑类(Imidazoles);脂多糖类(Lipopolysaccharides);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);膜蛋白质类(Membrane Proteins);线粒体蛋白质类(Mitochondrial Proteins);有机金属化合物(Organometallic Compounds);氧化性应激(Oxidative Stress);吡啶类(Pyridines);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);治疗结果(Treatment Outcome);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.jss.2018.03.042
PMID
29907213
发布时间
2020-03-16
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