Enhanced activation of interleukin-10, heme oxygenase-1, and AKT in C5aR2-deficient mice is associated with protection from ischemia reperfusion injury-induced inflammation and fibrosis.
作者:
主题词
动物(Animals);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);上皮细胞(Epithelial Cells);纤维化(Fibrosis);血红素加氧酶-1(Heme Oxygenase-1);炎症(Inflammation);白细胞介素10(Interleukin-10);肾(Kidney);肾小管(Kidney Tubules);脂多糖类(Lipopolysaccharides);巨噬细胞(Macrophages);磁共振成像(Magnetic Resonance Imaging);膜蛋白质类(Membrane Proteins);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);灌注成像(Perfusion Imaging);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);受体, 过敏毒素C5a(Receptor, Anaphylatoxin C5a);再生(Regeneration);再灌注损伤(Reperfusion Injury);增量调节(Up-Regulation)
DOI
10.1016/j.kint.2018.04.005
PMID
29935951
发布时间
2019-06-24
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Kidney international
741-755页
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