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首页> Genes & development> The C9orf72-interacting protein Smcr8 is a negative regulator of autoimmunity and lysosomal exocytosis.

The C9orf72-interacting protein Smcr8 is a negative regulator of autoimmunity and lysosomal exocytosis.

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作者： Yingying,Zhang ^[1] ; Aaron,Burberry ^[2] ; Jin-Yuan,Wang ^[3] ; Jackson,Sandoe ^[1] ; Sulagna,Ghosh ^[2] ; Namrata D,Udeshi ^[3] ; Tanya,Svinkina ^[1] ; Daniel A,Mordes ^[2] ; Joanie,Mok ^[3] ; Maura,Charlton ^[1] ; Quan-Zhen,Li ^[2] ; Steven A,Carr ^[3] ; Kevin,Eggan ^[1]

作者单位： Department of Stem Cell and Regenerative Biology, Harvard University, Cambridge, Massachusetts 02138, USA. ^[1] Department of Molecular and Cellular Biology, Harvard University, Cambridge, Massachusetts 02138, USA. ^[2] Stanley Center for Psychiatric Research, Broad Institute of Massachusetts Institute of Technology and Harvard, Cambridge, Massachusetts 02142, USA. ^[3] Proteomics Platform, Broad Institute of MIT and Harvard, Cambridge, Massachusetts 02142, USA. ^[4] Department of Pathology, Massachusetts General Hospital, Boston, Massachusetts 02114, USA. ^[5] Department of Immunology, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA. ^[6] Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA. ^[7]

关键词 C9ORF72 SMCR8 amyotrophic lateral sclerosis (ALS)autoimmunity autophagy lysosome

主题词肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);自身免疫(Autoimmunity);载体蛋白质类(Carrier Proteins);胞吐作用(Exocytosis);基因表达调控(Gene Expression Regulation);人类(Humans);淋巴结(Lymph Nodes);溶酶体相关膜蛋白质1(Lysosomal-Associated Membrane Protein 1);溶酶体(Lysosomes);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);突变(Mutation);蛋白质亚型(Protein Isoforms);蛋白质稳定性(Protein Stability);脾大(Splenomegaly)

DOI 10.1101/gad.313932.118

PMID 29950492

发布时间 2018-12-02

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Genes & development

Genes & development

2018年32卷13-14期

929-943页

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