Global phosphoproteomics of CCR5-tropic HIV-1 signaling reveals reprogramming of cellular protein production pathways and identifies p70-S6K1 and MK2 as HIV-responsive kinases required for optimal infection of CD4+ T cells.
作者:
主题词
CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);细胞骨架(Cytoskeleton);HIV感染(HIV Infections);HIV-1(HIV-1);宿主与病原体相互作用(Host-Pathogen Interactions);人类(Humans);免疫记忆(Immunologic Memory);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);磷蛋白类(Phosphoproteins);蛋白质组学(Proteomics);受体, CCR5(Receptors, CCR5);受体, CXCR4(Receptors, CXCR4);核糖体蛋白质S6激酶类, 70-kDa(Ribosomal Protein S6 Kinases, 70-kDa);信号传导(Signal Transduction);病毒嗜亲性(Viral Tropism);病毒复制(Virus Replication)
DOI
10.1186/s12977-018-0423-4
PMID
29970186
发布时间
2021-12-04
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