Blast exposure elicits blood-brain barrier disruption and repair mediated by tight junction integrity and nitric oxide dependent processes.
第一作者:
Aric F,Logsdon
第一单位:
Geriatric Research Education and Clinical Center (GRECC), Veterans Affairs Puget Sound Health Care System, Seattle, WA, 98108, USA.;Division of Gerontology and Geriatric Medicine, Department of Medicine, University of Washington School of Medicine, Seattle, WA, 98195, USA.
作者:
主题词
白蛋白类(Albumins);动物(Animals);爆震伤(Blast Injuries);血脑屏障(Blood-Brain Barrier);碳放射性同位素(Carbon Radioisotopes);环加氧酶抑制药(Cyclooxygenase Inhibitors);神经胶质增生(Gliosis);男(雄)性(Male);小鼠, 近交C57BL(Mice, Inbred C57BL);NG-硝基精氨酸甲酯(NG-Nitroarginine Methyl Ester);一氧化氮(Nitric Oxide);一氧化氮合酶(Nitric Oxide Synthase);器官特异性(Organ Specificity);通透性(Permeability);放射性药物(Radiopharmaceuticals);蔗糖(Sucrose);紧密连接部(Tight Junctions);时间因素(Time Factors);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1038/s41598-018-29341-6
PMID
30054495
发布时间
2019-10-16
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