Loss of histone deacetylase 2 inhibits oxidative stress induced by high glucose via the HO-1/SIRT1 pathway in endothelial progenitor cells.
第一作者:
Jie,Gao
第一单位:
Department of peripheral vascular disease, First Affiliated Hospital, Heilongjiang University of Chinese Medicine, Haerbin, 150040, Heilongjiang, China.
作者:
主题词
细胞增殖(Cell Proliferation);细胞因子类(Cytokines);糖尿病足(Diabetic Foot);葡萄糖(Glucose);血红素加氧酶-1(Heme Oxygenase-1);组蛋白脱乙酰基酶2(Histone Deacetylase 2);人类(Humans);氧化性应激(Oxidative Stress);RNA, 小分子干扰(RNA, Small Interfering);活性氧(Reactive Oxygen Species);信号传导(Signal Transduction);抗衰老酶1(Sirtuin 1);增量调节(Up-Regulation)
DOI
10.1016/j.gene.2018.07.072
PMID
30063937
发布时间
2018-10-22
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