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首页> Neurobiology of disease> Inactivation of Magel2 suppresses oxytocin neurons through synaptic excitation-inhibition imbalance.

Inactivation of Magel2 suppresses oxytocin neurons through synaptic excitation-inhibition imbalance.

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第一作者： Tayfun,Ates

第一单位： Department of Physiology, School of Medicine, Regenerative and Restorative Medical Research Center (REMER), Istanbul Medipol University, Istanbul, Turkey.

作者： Tayfun,Ates ^[1] ; Merve,Oncul ^[1] ; Pelin,Dilsiz ^[1] ; Iskalen Cansu,Topcu ^[2] ; Cihan Civan,Civas ^[2] ; Muhammed Ikbal,Alp ^[1] ; Iltan,Aklan ^[2] ; Edanur,Ates Oz ^[1] ; Yavuz,Yavuz ^[2] ; Bayram,Yilmaz ^[2] ; Nilufer,Sayar Atasoy ^[1] ; Deniz,Atasoy ^[3]

作者单位： Department of Physiology, School of Medicine, Regenerative and Restorative Medical Research Center (REMER), Istanbul Medipol University, Istanbul, Turkey. ^[1] Department of Physiology, School of Medicine, Yeditepe University, Istanbul, Turkey. ^[2] Department of Physiology, School of Medicine, Regenerative and Restorative Medical Research Center (REMER), Istanbul Medipol University, Istanbul, Turkey. Electronic address: datasoy@medipol.edu.tr. ^[3]

关键词 AMPA Autism Electrophysiology NMDA Oxytocin Prader Willi Syndrome magel2

主题词动作电位(Action Potentials);动物(Animals);抗原, 肿瘤(Antigens, Neoplasm);兴奋性突触后电位(Excitatory Postsynaptic Potentials);女(雌)性(Female);下丘脑(Hypothalamus);抑制性突触后电位(Inhibitory Postsynaptic Potentials);男(雄)性(Male);膜电位(Membrane Potentials);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);神经元(Neurons);催产素(Oxytocin);蛋白质类(Proteins);受体, AMPA(Receptors, AMPA)

DOI 10.1016/j.nbd.2018.09.017

PMID 30240706

发布时间 2019-11-21

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