换一批USP49 inhibits ischemia-reperfusion-induced cell viability suppression and apoptosis in human AC16 cardiomyocytes through DUSP1-JNK1/2 signaling.
第一作者：
Wei,Zhang
第一单位：
Department of Cardiology, Shanghai Tenth People's Hospital, Tongji University School of Medicine, Shanghai, China.
作者：
主题词
动物(Animals);细胞凋亡(Apoptosis);细胞存活(Cell Survival);双特异性磷酸酶1(Dual Specificity Phosphatase 1);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);MAP激酶信号系统(MAP Kinase Signaling System);丝裂原活化蛋白激酶8(Mitogen-Activated Protein Kinase 8);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);肌细胞, 心脏(Myocytes, Cardiac);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);再灌注损伤(Reperfusion Injury);信号传导(Signal Transduction);泛素硫酯酶(Ubiquitin Thiolesterase);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1002/jcp.27390
PMID
30246457
发布时间
2020-04-08
- 浏览6
Journal of cellular physiology
6529-6538页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
