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Rapamycin and fasting sustain autophagy response activated by ischemia/reperfusion injury and promote retinal ganglion cell survival.

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第一作者: Rossella,Russo
第一单位: Department of Pharmacy, Health and Nutritional Sciences, Section of Preclinical and Translational Pharmacology, University of Calabria, 87036, Arcavacata di Rende, Italy. rossella.russo@unical.it.
作者单位: Department of Pharmacy, Health and Nutritional Sciences, Section of Preclinical and Translational Pharmacology, University of Calabria, 87036, Arcavacata di Rende, Italy. rossella.russo@unical.it. [1] Department of Pharmacy, Health and Nutritional Sciences, Section of Preclinical and Translational Pharmacology, University of Calabria, 87036, Arcavacata di Rende, Italy.;Ophtalmology Unit, Department of Experimental Medicine, University of Rome "Tor Vergata", 00133, Rome, Italy. [2] Department of Pharmacy, Health and Nutritional Sciences, Section of Preclinical and Translational Pharmacology, University of Calabria, 87036, Arcavacata di Rende, Italy. [3] Department of Pediatric Hematology and Oncology, IRCSS Bambino Gesù Children's Hospital, Rome, Italy. [4] Department of Biotechnology and Life Sciences, University of Insubria, 21100, Varese, Italy. [5] Cell Stress and Survival Unit, Center for Autophagy, Recycling and Disease (CARD), Danish Cancer Society Research Center, 2100, Copenhagen, Denmark. [6] Department of Health Sciences, University "Magna Graecia" of Catanzaro, 88100, Catanzaro, Italy. [7] Ophtalmology Unit, Department of Experimental Medicine, University of Rome "Tor Vergata", 00133, Rome, Italy. [8]
DOI 10.1038/s41419-018-1044-5
PMID 30250019
发布时间 2022-04-09
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