CCL18 secreted from M2 macrophages promotes migration and invasion via the PI3K/Akt pathway in gallbladder cancer.

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作者： Zhenyu,Zhou ^[1] ; Yaorong,Peng ^[2] ; Xiaoying,Wu ^[1] ; Shiyu,Meng ^[3] ; Wei,Yu ^[4] ; Jinghua,Zhao ^[1] ; Heyun,Zhang ^[5] ; Jie,Wang ^[1] ; Wenbin,Li ^[2]

作者单位： Guangdong Province Key Laboratory of Malignant Tumor Epigenetics and Gene Regulation, Research Center of Medicine, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, 510120, China. ^[1] Department of Hepatobiliary Surgery, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, No. 33 Yingfeng Road, Guangzhou, 510289, China. ^[2] Department of Pancreaticobiliary Surgery, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, No. 33 Yingfeng Road, Guangzhou, 510289, China. ^[3] Department of General Surgery, The Seventh Affiliated Hospital, Sun Yat-Sen University, Shenzhen, 518107, China. ^[4] Department of Anesthesiology, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, 510289, China. ^[5] Breast Tumor Center, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, Guangzhou, 510120, China. ^[6] Department of Hepatobiliary Surgery, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, No. 33 Yingfeng Road, Guangzhou, 510289, China. jiewsysu@163.com. ^[7] Department of Pancreaticobiliary Surgery, Sun Yat-Sen Memorial Hospital, Sun Yat-Sen University, No. 33 Yingfeng Road, Guangzhou, 510289, China. gzsurgeon@163.com. ^[8]

关键词 CCL18 Chronic inflammation Epithelial-mesenchymal transition Gallbladder cancer M2 macrophages