XBP1-KLF9 Axis Acts as a Molecular Rheostat to Control the Transition from Adaptive to Cytotoxic Unfolded Protein Response.

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作者： Emily E,Fink ^[1] ; Sudha,Moparthy ^[2] ; Archis,Bagati ^[1] ; Anna,Bianchi-Smiraglia ^[1] ; Brittany C,Lipchick ^[2] ; David W,Wolff ^[2] ; Matthew V,Roll ^[2] ; Jianmin,Wang ^[3] ; Song,Liu ^[3] ; Andrei V,Bakin ^[4] ; Eugene S,Kandel ^[1] ; Ann-Hwee,Lee ^[5] ; Mikhail A,Nikiforov ^[6]

作者单位： Department of Cell Stress Biology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA. ^[1] Department of Cell Stress Biology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA; Department of Cancer Biology, Comprehensive Cancer Center of Wake Forest Baptist Medical Center, Winston-Salem, NC 27157, USA. ^[2] Department of Biostatistics and Bioinformatics, Roswell Park Cancer Institute, Buffalo, NY 14263, USA. ^[3] Department of Cancer Genetics and Genomics, Roswell Park Cancer Institute, Buffalo, NY 14263, USA. ^[4] Department of Pathology and Laboratory Medicine, Weill Cornell Medical College, New York, NY 10065, USA. ^[5] Department of Cell Stress Biology, Roswell Park Cancer Institute, Buffalo, NY 14263, USA; Department of Cancer Biology, Comprehensive Cancer Center of Wake Forest Baptist Medical Center, Winston-Salem, NC 27157, USA. Electronic address: mnikifor@wakehealth.edu. ^[6]

关键词 ITPR1 KLF9 TMEM38B UPR XBP1s calcium channel endoplasmic reticulum stress