TWEAK promotes endothelial progenitor cell vasculogenesis to alleviate acute myocardial infarction via the Fn14-NF-κB signaling pathway.

导出 LinkOut

翻译打印收藏纠错

第一作者： Zulong,Sheng

第一单位： Department of Cardiology, Zhongda Hospital, Medical School of Southeast University, Nanjing, Jiangsu 210009, P.R. China.

作者： Zulong,Sheng ^[1] ; Chenwei,Ju ^[1] ; Bing,Li ^[1] ; Zhongpu,Chen ^[1] ; Xiaodong,Pan ^[1] ; Gaoliang,Yan ^[1] ; Yanru,He ^[1] ; Yuyu,Yao ^[1] ; Genshan,Ma ^[1]

作者单位： Department of Cardiology, Zhongda Hospital, Medical School of Southeast University, Nanjing, Jiangsu 210009, P.R. China. ^[1]

关键词 acute myocardial infarction endothelial progenitor cells nuclear factor-κB signal tumor necrosis factor-related weak inducer of apoptosis vasculogenesis

DOI 10.3892/etm.2018.6703

PMID 30344680

发布时间 2020-09-29

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览6

Experimental and therapeutic medicine

2018年16卷5期

4019-4029页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

TWEAK promotes endothelial progenitor cell vasculogenesis to alleviate acute myocardial infarction via the Fn14-NF-κB signaling pathway.

Experimental and therapeutic medicine

相似文献

Experimental and therapeutic medicine

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

TWEAK promotes endothelial progenitor cell vasculogenesis to alleviate acute myocardial infarction via the Fn14-NF-κB signaling pathway.

Experimental and therapeutic medicine

相似文献

Experimental and therapeutic medicine

相关期刊

检索历史清除